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Manganese and inflammation
Still reacting negatively to all foods except meat and some rice.
Recently I've tried nuts and got a dementia like brain fog for like 12 hours straight.
I've looked into what nuts contain and among other things like lectins, etc its also a rich source of manganese, so what's so special about this trace element, lets see...
Microglia amplify inflammatory activation of astrocytes in manganese neurotoxicity:
https://jneuroinflammation.biomedcentral.com/articles/10.1186/s12974-017-0871-0
Pro-inflammatory cytokine and vascular adhesion molecule levels in manganese and lead-exposed workers:
https://www.peertechzpublications.com/articles/IJICR-5-120.php
Manganese Potentiates In Vitro Production of Proinflammatory Cytokines and Nitric Oxide by Microglia Through a Nuclear Factor kappa B–Dependent Mechanism:
https://academic.oup.com/toxsci/article/84/1/139/1610857
Some examples of foods that are good sources of manganese include:
nuts, such as almonds and pecans.
beans and legumes, such as lima and pinto beans.
oatmeal and bran cereals.
whole wheat bread.
brown rice.
leafy green vegetables, such as spinach.
fruits, such as pineapple and acai.
dark chocolate.
If all you're eating is beef and some rice you might be "deficient" in polyunsaturated fat, which is a good state to be in because then your body produces a lot of the anti-inflammatory omega-9 mead acid. Eating any kind of nut except for macadamias would quickly kick you of of that """deficiency""" state, for me that results in much worsened brainfog and cognitive function. Just a thought. Although this is probably not the culprit if it only takes you 12 hours to fully recover.
White rice also has plenty of manganese.
Just a thought for you to look into. Free glutamate.
+++
lectins could promote inflammation too:
Plant Lectins Activate the NLRP3 Inflammasome To Promote Inflammatory Disorders:
https://www.jimmunol.org/content/198/5/2082
Although we demonstrate that plant lectins can induce strong NLRP3 inflammasome activation in macrophages, the concentration needed for NLRP3 activation in vitro is high. Indeed, the physiological concentration of lectins should be much lower, because the intestinal barrier could prevent the entry of lectins to the body (54). However, in patients with inflammatory bowel disease, the concentrations of serum WGA Abs are much higher than in healthy controls (37), suggesting that more lectins can enter the body and trigger NLRP3-dependent inflammation when the intestinal barrier is disrupted. Additionally, the physiological concentration of lectins might trigger low-grade and persistent NLRP3-dependent inflammation, which then contributes to chronic inflammatory diseases.