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Quote from Janelle525 on August 1, 2024, 7:13 amQuote from tim on August 1, 2024, 3:03 am@janelle525
So this is interesting... blindness from an apparent fat soluble deficiency occurs because of inflammation from a pathogen because it destroys the glands and ducts that are required to keep the eye moist.
The pathogen component of the mechanism of causation could explain, if some are more susceptible than others, why.
Perhaps retinoic acid deficiency doesn't actually cause a significant reduction in lacrimal fluid directly. Lacrimal fluid contains various anti bacterial proteins, if production of those were affected then that could explain lack of observable pathology until infection occurred.
@tim-2, yeah it appears that is what they found. It seems the lungs also were infected. The thymus was destroyed as well. The liver and intestines were normal though. So what kills someone in fat soluble deficiency appears to be infection. She had a fever.
Karen Hurd explained that with dry mucous membranes there is a fat deficiency and to add more nuts and oils to the diet (she thinks saturated fat is harmful), but nuts and oils don't have retinol.
When I was researching dry eyes, it is usually because of meibomian gland dysfunction, these glands produce oil that keeps our eyes from evaporating the tears. They also found that demodex mites could be the cause of the dysfunction. I wonder what factors help us fight off the mites? Because obviously not everyone has this issue. Does retinoic acid help kill bacteria and mites? I must have enough retinoic acid though because I haven't gone longer than a week or two without dairy fat and I eat beef fat everyday. Salt I think is a factor for me. Water follows salt. In a baby only consuming 10 ounces of diluted skim milk a day I could see dehydration being a major factor in infection. The skin was waxy and inelastic. This was right before penicillin was invented, had they had antibiotics this baby (and the others) might still have survived.
Quote from tim on August 1, 2024, 3:03 amSo this is interesting... blindness from an apparent fat soluble deficiency occurs because of inflammation from a pathogen because it destroys the glands and ducts that are required to keep the eye moist.
The pathogen component of the mechanism of causation could explain, if some are more susceptible than others, why.
Perhaps retinoic acid deficiency doesn't actually cause a significant reduction in lacrimal fluid directly. Lacrimal fluid contains various anti bacterial proteins, if production of those were affected then that could explain lack of observable pathology until infection occurred.
@tim-2, yeah it appears that is what they found. It seems the lungs also were infected. The thymus was destroyed as well. The liver and intestines were normal though. So what kills someone in fat soluble deficiency appears to be infection. She had a fever.
Karen Hurd explained that with dry mucous membranes there is a fat deficiency and to add more nuts and oils to the diet (she thinks saturated fat is harmful), but nuts and oils don't have retinol.
When I was researching dry eyes, it is usually because of meibomian gland dysfunction, these glands produce oil that keeps our eyes from evaporating the tears. They also found that demodex mites could be the cause of the dysfunction. I wonder what factors help us fight off the mites? Because obviously not everyone has this issue. Does retinoic acid help kill bacteria and mites? I must have enough retinoic acid though because I haven't gone longer than a week or two without dairy fat and I eat beef fat everyday. Salt I think is a factor for me. Water follows salt. In a baby only consuming 10 ounces of diluted skim milk a day I could see dehydration being a major factor in infection. The skin was waxy and inelastic. This was right before penicillin was invented, had they had antibiotics this baby (and the others) might still have survived.
Quote from Janelle525 on August 1, 2024, 7:47 am@jessica2
This kinda confirms a theory I had that people with low blood pressure for yrs and yrs go on to develop hyperaldosteronism and high blood pressure and insulin problems because yrs and yrs of the kidneys calling on the adrenal glands to conserve sodium results in disease. So doctors who don't know crap telling people oh awesome your blood pressure is low! is harming thousands of people. Kidneys are salt filtering organs not salt conserving organs, they have to call upon adrenal hormones to conserve sodium, which then wastes potassium which causes the diabetes and high blood pressure. So my lower salt diet for the last 6 months could explain my worsening fasting glucose.
@jessica2
This kinda confirms a theory I had that people with low blood pressure for yrs and yrs go on to develop hyperaldosteronism and high blood pressure and insulin problems because yrs and yrs of the kidneys calling on the adrenal glands to conserve sodium results in disease. So doctors who don't know crap telling people oh awesome your blood pressure is low! is harming thousands of people. Kidneys are salt filtering organs not salt conserving organs, they have to call upon adrenal hormones to conserve sodium, which then wastes potassium which causes the diabetes and high blood pressure. So my lower salt diet for the last 6 months could explain my worsening fasting glucose.
Quote from Viktor2 on August 2, 2024, 11:06 pm@janelle at the beginning of my low VA journey I had quite bad dry eyes, probably just a detox side effect. Now in summer where I eat more loosely I'm seeing some again. I don't think it's from deficiency.
@janelle at the beginning of my low VA journey I had quite bad dry eyes, probably just a detox side effect. Now in summer where I eat more loosely I'm seeing some again. I don't think it's from deficiency.
Quote from Janelle525 on August 3, 2024, 8:25 am@viktor2, yeah I don't think it's a sign of deficiency either.
@viktor2, yeah I don't think it's a sign of deficiency either.