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Sun exposure
Do you guys have also more intense detox symptoms when you have a lot of sun exposure?
Since 1 month I get daily a good amount of sun and react good and badly.
I see a lot of progress but when I spend too much time in the sun, it becomes pretty intensive.
I've been meaning to write about my latest sun exposure experience. Up till July 2nd I stayed away from sun exposure because last summer I got a terrible foot rash that prevented me from being very mobile or from sleeping well and for this summer, I didn't want that to ruin a vacation to Orlando where I would allow significant sun exposure. This past July 2nd to the 7th while vacationing in Orlando I was outside in the mornings till around half past noon - not outside in the sun the whole mornings but in out of shade so don't know how much time in the sun. I avoided being outside from around 12:30 to 4pm on those 6 days. On my last day in Florida, I spent much of the afternoon outside in the sun between noon and 3:30. That night I flew home and the next day and everday since I have been very fatigued. It wasn't the vacation - I didn't overdo anything except probably sun exposure. I sleep around 8 hours now a night and then usually have to take a nap or 2. I am also getting those unique toxic vA headaches. While in Florida, I didn't need that much sleep and didn't have headaches. Either the morning sun exposure didn't affect me or didn't affect me till I got home? I think what really did me in was that last day since I didn't seek shade that much the afternoon I was outside. Anyway it has been about 2 weeks of this feeling very tired. I have mostly avoided the sun here at home in Colordado though I get outside in the morning sometimes around 8 or 9 to do yard work for an hour or so or take a walk in mostly shaded areas. I know the sun is healing but I think it is making me feel really ill right now because of detoxing. Luckily I did not get a bad rash this summer - had a slight one on my feet while in Florida, but it seemed more like a heat rash that stopped itching after I would take off my shoes and socks.
At the beginning I was considering quick daily sun baths as an enjoyable part of the therapy.
After a while, I got the intuition that was a bit intensive.
Now, I'm doing it only sometimes, when it feels smart. Also, I'm not worrying too much when life sends me into the sun for short periods.
Maybe sun is good for people who feel they are at a plateau.
I'm starting to think that sun might be like alcohol. And perhaps like getting the flu, bug bites, infections, harsh heat and cold.
It stirs the pot.
My guess is that if you checked your circulating A on days when you do things like that list...it's there in higher concentrations. Getting used up.
Perhaps one of the issues (I can see it with myself) is that modern life can sometimes be "not challenging enough" for us to use up VA.
I also want to say there might be a strange look to VA overload people's TANS... something I've noticed.
If vit A antagonizes(modulates) vit D bio-availability, increased sun intolerance on a low A makes sense
https://ggenereux.blog/discussion/topic/vitamin-a-antagonizes-vitamin-d/
Anyone else noticed _increased_ sun sensitivity(like fatigue after sun exposure) on a low vit A diet?
@rockarolla, it seems to me that if my skin hasn't been exposed to the sun in a while I will come out itchy in the first few outings. Best to get that over with before a fun vacation. After that initial phase I'm a bit more sun tolerant than I was before.
I can only take so much sun exposure when it comes to my eyes, even though they get light all year round. It can make you feel like crawling into a hole if too much of that happens.
I'm not very pigmented, though.
I'am now much more sensitive to the sun compared to the previous year when sun had only a cooling effect on all of my symptoms leading to the temporal addiction, and a huge rollback at the autumn.
These days on a very low A diet the sun knocks me into the 2..3 chunks several hours apart of 4..6 hour sleep(narcolepsia) 1..2 hours after exposure and I do not feel any anti inflammatory effect from it.
I have a theory that liver-stored vitamin A is used by the body as an antidote(antagonist or a weak agonist) to too much stimulation of vitamin D receptors via calcitriol - could explain why milk contains A but not D(to protect the baby from accidental D overstimulation by preoccupying receptors with A). Also explains the mobilisation of A(sick person context only) through sunbathing.
As for adults the protection is probably always excessive leading to the chronic immune system down regulation especially with every day A intake, temporary masking symptoms of the disease, and eventually a regression.
Riboflavin is very sensitive to sunlight. I don't know how much is lost during sun exposure but it's well understood that folate is depleted during sun exposure.
Abstract
Vitamin D is unique in being generated in our skin following ultraviolet radiation (UVR) exposure. Ongoing research into vitamin D must therefore always consider the influence of UVR on vitamin D processes. The close relationship between vitamin D and UVR forms the basis of the “vitamin D–folate hypothesis”, a popular theory for why human skin colour has evolved as an apparent adaption to UVR environments. Vitamin D and folate have disparate sensitivities to UVR; whilst vitamin D may be synthesised following UVR exposure, folate may be degraded. The vitamin D–folate hypothesis proposes that skin pigmentation has evolved as a balancing mechanism, maintaining levels of these vitamins. There are several alternative theories that counter the vitamin D–folate hypothesis. However, there is significant overlap between these theories and the now known actions of vitamin D and folate in the skin. The focus of this review is to present an update on the vitamin D–folate hypothesis by integrating these current theories and discussing new evidence that supports associations between vitamin D and folate genetics, UVR, and skin pigmentation. In light of recent human migrations and seasonality in disease, the need for ongoing research into potential UVR-responsive processes within the body is also discussed.
I feel that I'm significantly more resistant to the sun after being on a low vitamin A diet for over two years. Two reason people might not see that benefit is if their retinoic acid levels are still high or they are deficient in other protective nutrients.
Photodecomposition and Phototoxicity of Natural Retinoids
Perspective
Regarding human toxicity, the long-term consequences of using cosmetics containing RP are currently unknown. It has been demonstrated that photoirradiation of RP can result in forming toxic photodecomposition products, generate ROS, induce lipid peroxidation, and cause DNA damage. Also, topically applied RP produces many of the cutaneous changes associated with the use of drug products containing RA which in some instances can enhance photocarcinogenesis. Thus, a study of the photocarcinogenesis of RP, under conditions relevant to the use of RP in cosmetics, is timely and important. As a consequence, RP has recently been nominated by the U.S. FDA and selected by the National Toxicology Program (NTP) as a high priority compound for phototoxicity and photocarcinogenicity studies. The goal of these studies is to provide relevant information necessary for risk assessment of RP in cosmetic creams.
My D25 is very low - last time I've checked it was only 10 ng ;(
Maybe I'am more sensitive to the sun due to less D25 blocking VDR(if D25 is indeed a VDR antagonist):
Oral Vitamin D Rapidly Attenuates Inflammation from Sunburn: An Interventional Study
The diverse immunomodulatory effects of vitamin D are increasingly being recognized. However, the ability of oral vitamin D to modulate acute inflammation in vivo has not been established in humans. In a double-blinded, placebo-controlled interventional trial, 20 healthy adults were randomized to receive either placebo or a high dose of vitamin D3 (cholecalciferol) one hour after experimental sunburn induced by an erythemogenic dose of UVR. Compared with placebo, participants receiving vitamin D3 (200,000 international units) demonstrated reduced expression of proinflammatory mediators tumor necrosis factor-α (P = 0.04) and inducible nitric oxide synthase (P = 0.02) in skin biopsy specimens 48 hours after experimental sunburn. A blinded, unsupervised hierarchical clustering of participants based on global gene expression profiles revealed that participants with significantly higher serum vitamin D3 levels after treatment (P = 0.007) demonstrated increased skin expression of the anti-inflammatory mediator arginase-1 (P = 0.005), and a sustained reduction in skin redness (P = 0.02), correlating with significant expression of genes related to skin barrier repair. In contrast, participants with lower serum vitamin D3 levels had significant expression of proinflammatory genes. Together the data may have broad implications for the immunotherapeutic properties of vitamin D in skin homeostasis, and implicate arginase-1 upregulation as a previously unreported mechanism by which vitamin D exerts anti-inflammatory effects in humans.