I needed to disable self sign-ups because I’ve been getting too many spam-type accounts. Thanks.
Anyone else craving fatty foods?
Lately been craving(and eating) a lot of fat. It started last week with severe cravings for heavy cream, peanut butter, fried foods, fatty beef, beef marrow, and a few other things. Additionally, starchy foods are not as appealing, and sugary foods are even less appealing. The only sweet foods I am continuing to eat is some fruit and small amounts of sugar. Over Christmas, there was ice cream, cookies, pies and cakes, but I could hardly indulge. One bite of a small cookie(no butter or eggs in these cookies) would even leave me filling a little nauseous.
I don't know what to say about it, and it seems a little abnormal. The heavy cream did cause a rash to break out around my stomach, but that has dried out and healed as I continued to eat more fat. I am eating something like 200 grams of fat a day at this point. I can't say if it's good or not, but the craving is very clear.
I also want to make clear that I've been getting quite a bit of preformed Vitamin A with the heavy cream(about 3/4 a cup a day) this last week. I don't recommend this. I am certain this change has caused some rashes to reappear. But I am continuing to eat fat, because I do crave it. Additionally, my mood has continued to be stable and I've been having no spinal pain.
Has anyone else been having this experience?
The diet is pretty low in fat so maybe your body just really wanted more fat. Or maybe you need the calcium. Parsnip has quite a lot of calcium and has no vitamin A. Cream is just about the single worst fat you could eat in terms of vitamin A. Or wait butter is even worse actually, I just looked it up. Peanut butter/oil and coconut oil have almost no vitamin A but some people have issues with them anyways but they can hardly be worse than cream. Processed and deodorized vegetable oils have quite low amounts of vitamin A as well, but if they are yellow they have carotenoids.
It makes sense that dairy fats are calming, at least initially, it has tons of progesterone in it. It binds to GABA receptors but it's also quite feminizing so if you're a guy you might want to reconsider that.
I had a stretch where I was craving more fat than usual. I mostly ate macadamia nuts and put lots of olive oil on my rice/beans/meat combo.
Bella
I've been thinking there is more to the story. I've done some reading, and apparently carbohydrate restricted diets reduce Retinol Binding Protein in comparison to calorically restricted high carbohydrate diets. When I get back home, I'll post a few studies in the other section. Additionally, I think low carbohydrate may reduce viral load, thus lowering the need for releasing Vitamin A into the serum
This could explain why I've been having a reduced craving for sugar and carbohydrates in general. The honey whole wheat bread I've been eating has even been tasting far too sweet for me. I'm still eating carbs, just far less and to appetite.
Quote from YH on December 27, 2018, 5:51 pmI've been thinking there is more to the story. I've done some reading, and apparently carbohydrate restricted diets reduce Retinol Binding Protein in comparison to calorically restricted high carbohydrate diets. When I get back home, I'll post a few studies in the other section. Additionally, I think low carbohydrate may reduce viral load, thus lowering the need for releasing Vitamin A into the serum
This could explain why I've been having a reduced craving for sugar and carbohydrates in general. The honey whole wheat bread I've been eating has even been tasting far too sweet for me. I'm still eating carbs, just far less and to appetite.
Interesting, I’m looking forward to reading the studies. I’m not eating or craving as much sugar now that I think about it...
Quote from YH on December 27, 2018, 5:51 pmI've been thinking there is more to the story. I've done some reading, and apparently carbohydrate restricted diets reduce Retinol Binding Protein in comparison to calorically restricted high carbohydrate diets. When I get back home, I'll post a few studies in the other section. Additionally, I think low carbohydrate may reduce viral load, thus lowering the need for releasing Vitamin A into the serum
This could explain why I've been having a reduced craving for sugar and carbohydrates in general. The honey whole wheat bread I've been eating has even been tasting far too sweet for me. I'm still eating carbs, just far less and to appetite.
You want more retinol binding protein, not less... It's what helps you detox retinol from the liver and get it out of your body. At least that's what Garrett Smith says. Not every craving is related to vitamin A status.
Quote from Guest on December 27, 2018, 8:57 pmQuote from YH on December 27, 2018, 5:51 pmI've been thinking there is more to the story. I've done some reading, and apparently carbohydrate restricted diets reduce Retinol Binding Protein in comparison to calorically restricted high carbohydrate diets. When I get back home, I'll post a few studies in the other section. Additionally, I think low carbohydrate may reduce viral load, thus lowering the need for releasing Vitamin A into the serum
This could explain why I've been having a reduced craving for sugar and carbohydrates in general. The honey whole wheat bread I've been eating has even been tasting far too sweet for me. I'm still eating carbs, just far less and to appetite.
You want more retinol binding protein, not less... It's what helps you detox retinol from the liver and get it out of your body. At least that's what Garrett Smith says. Not every craving is related to vitamin A status.
I think Garrett is wrong about this. Assuming a normal diet, RBP is produced in response to retinol ingestion, or even injection. Additionally, the level of Vitamin A and RBP in your body is determined by a number of other factors besides how much is ingested. Eating zinc and protein will not simply make more RPB. In fact RPB is produced independent of protein intake. I posted a study about this in the other section.
Plasma retinol-binding protein.
Abstract
Vitamin A is mobilized from liver stores and transported in plasma in the form of the lipid alcohol retinol, bound to a specific transport protein, retinol-binding protein (RBP). A great deal is known about the chemical structure, metabolism, and biological roles of RBP. RBP is a single polypeptide chain with molecular weight close to 20,000. RBP interacts strongly with plasma prealbumin, and normally circulates in plasma as a 1:1 molar RBP-prealbumin complex. Both the primary and the tertiary structure of prealbumin are known, and the primary structure of RBP has recently been reported. Much information is available about the protein-protein and protein-ligand interactions that are involved in this transport system. Many clinical studies have examined the effects of a variety of diseases on the plasma levels of RBP and prealbumin in humans. Plasma RBP levels are low in patients with liver disease and are high in patients with chronic renal disease. These findings reflect the facts that RBP is produced in the liver and mainly catabolized in the kidneys. Delivery of retinol to extra-hepatic tissues appears to involve specific cell surface receptors for RBP. Vitamin A mobilization from the liver, and delivery to peripheral tissues, is highly regulated by factors that control the rates of RBP production and secretion. Retinol deficiency specifically blocks the secretion of RBP, so that plasma RBP levels fall and liver RBP levels rise. Injection of retinol into vitamin A-deficient rats stimulates the rapid secretion of RBP from the liver into the plasma. The cellular and molecular mechanisms that mediate these phenomena are under investigation. Elucidation of these mechanisms should help define the basic mechanisms that control the mobilization, transport, and delivery of vitamin A.
Quote from YH on December 27, 2018, 9:28 pmQuote from Guest on December 27, 2018, 8:57 pmQuote from YH on December 27, 2018, 5:51 pmI've been thinking there is more to the story. I've done some reading, and apparently carbohydrate restricted diets reduce Retinol Binding Protein in comparison to calorically restricted high carbohydrate diets. When I get back home, I'll post a few studies in the other section. Additionally, I think low carbohydrate may reduce viral load, thus lowering the need for releasing Vitamin A into the serum
This could explain why I've been having a reduced craving for sugar and carbohydrates in general. The honey whole wheat bread I've been eating has even been tasting far too sweet for me. I'm still eating carbs, just far less and to appetite.
You want more retinol binding protein, not less... It's what helps you detox retinol from the liver and get it out of your body. At least that's what Garrett Smith says. Not every craving is related to vitamin A status.
I think Garrett is wrong about this. Assuming a normal diet, RBP is produced in response to retinol ingestion, or even injection. Additionally, the level of Vitamin A and RBP in your body is determined by a number of other factors besides how much is ingested. Eating zinc and protein will not simply make more RPB. In fact RPB is produced independent of protein intake. I posted a study about this in the other section.
Plasma retinol-binding protein.
Abstract
Vitamin A is mobilized from liver stores and transported in plasma in the form of the lipid alcohol retinol, bound to a specific transport protein, retinol-binding protein (RBP). A great deal is known about the chemical structure, metabolism, and biological roles of RBP. RBP is a single polypeptide chain with molecular weight close to 20,000. RBP interacts strongly with plasma prealbumin, and normally circulates in plasma as a 1:1 molar RBP-prealbumin complex. Both the primary and the tertiary structure of prealbumin are known, and the primary structure of RBP has recently been reported. Much information is available about the protein-protein and protein-ligand interactions that are involved in this transport system. Many clinical studies have examined the effects of a variety of diseases on the plasma levels of RBP and prealbumin in humans. Plasma RBP levels are low in patients with liver disease and are high in patients with chronic renal disease. These findings reflect the facts that RBP is produced in the liver and mainly catabolized in the kidneys. Delivery of retinol to extra-hepatic tissues appears to involve specific cell surface receptors for RBP. Vitamin A mobilization from the liver, and delivery to peripheral tissues, is highly regulated by factors that control the rates of RBP production and secretion. Retinol deficiency specifically blocks the secretion of RBP, so that plasma RBP levels fall and liver RBP levels rise. Injection of retinol into vitamin A-deficient rats stimulates the rapid secretion of RBP from the liver into the plasma. The cellular and molecular mechanisms that mediate these phenomena are under investigation. Elucidation of these mechanisms should help define the basic mechanisms that control the mobilization, transport, and delivery of vitamin A.
If someone commits a crime and the police comes, the police isn't the problem. They are just responding to a problem and I'd rather they come than they don't. What happens if you inject retinol and your body is unable to produce retinol binding protein, doesn't the retinol just float about freely, wreaking whatever havoc it can?
Quote from Guest on December 28, 2018, 3:51 amQuote from YH on December 27, 2018, 9:28 pmQuote from Guest on December 27, 2018, 8:57 pmQuote from YH on December 27, 2018, 5:51 pmI've been thinking there is more to the story. I've done some reading, and apparently carbohydrate restricted diets reduce Retinol Binding Protein in comparison to calorically restricted high carbohydrate diets. When I get back home, I'll post a few studies in the other section. Additionally, I think low carbohydrate may reduce viral load, thus lowering the need for releasing Vitamin A into the serum
This could explain why I've been having a reduced craving for sugar and carbohydrates in general. The honey whole wheat bread I've been eating has even been tasting far too sweet for me. I'm still eating carbs, just far less and to appetite.
You want more retinol binding protein, not less... It's what helps you detox retinol from the liver and get it out of your body. At least that's what Garrett Smith says. Not every craving is related to vitamin A status.
I think Garrett is wrong about this. Assuming a normal diet, RBP is produced in response to retinol ingestion, or even injection. Additionally, the level of Vitamin A and RBP in your body is determined by a number of other factors besides how much is ingested. Eating zinc and protein will not simply make more RPB. In fact RPB is produced independent of protein intake. I posted a study about this in the other section.
Plasma retinol-binding protein.
Abstract
Vitamin A is mobilized from liver stores and transported in plasma in the form of the lipid alcohol retinol, bound to a specific transport protein, retinol-binding protein (RBP). A great deal is known about the chemical structure, metabolism, and biological roles of RBP. RBP is a single polypeptide chain with molecular weight close to 20,000. RBP interacts strongly with plasma prealbumin, and normally circulates in plasma as a 1:1 molar RBP-prealbumin complex. Both the primary and the tertiary structure of prealbumin are known, and the primary structure of RBP has recently been reported. Much information is available about the protein-protein and protein-ligand interactions that are involved in this transport system. Many clinical studies have examined the effects of a variety of diseases on the plasma levels of RBP and prealbumin in humans. Plasma RBP levels are low in patients with liver disease and are high in patients with chronic renal disease. These findings reflect the facts that RBP is produced in the liver and mainly catabolized in the kidneys. Delivery of retinol to extra-hepatic tissues appears to involve specific cell surface receptors for RBP. Vitamin A mobilization from the liver, and delivery to peripheral tissues, is highly regulated by factors that control the rates of RBP production and secretion. Retinol deficiency specifically blocks the secretion of RBP, so that plasma RBP levels fall and liver RBP levels rise. Injection of retinol into vitamin A-deficient rats stimulates the rapid secretion of RBP from the liver into the plasma. The cellular and molecular mechanisms that mediate these phenomena are under investigation. Elucidation of these mechanisms should help define the basic mechanisms that control the mobilization, transport, and delivery of vitamin A.
If someone commits a crime and the police comes, the police isn't the problem. They are just responding to a problem and I'd rather they come than they don't. What happens if you inject retinol and your body is unable to produce retinol binding protein, doesn't the retinol just float about freely, wreaking whatever havoc it can?
What I am saying is that Retinol Binding Protein is made in response to retinol consumption and injection. Additionally, retinol binding protein is released into the body independently of protein intake. You produce RPB even in a fasted state. Vitamin A is not a "criminal," it is a weapon. If the body is able to use it properly, it serves a valuable function, especially when it comes to infection and viruses. If it used improperly, it leads to autoimmunity and degenerative diseases. Additionally, excessive consumption of preformed or pure retonic acid could weigh against the bodies ability to produce retinol binding protein. In response, the body with produce more fatty tissue to deal with the excess retinol.
At the same time, the body will only keep the retinol if it needs it. Retinoic acid is excreted by the body if is not needed and if there is an excess supply that is arriving. If this system is working properly, there shouldn't be a problem. An excess consumption of preformed Vitamin A, whether with a supplement, Liver, or cold water fish could exasperate the body's ability to deal with Vitamin A.
This assumes “vitamin”-A is actually useful. Although it is an n=1 experiment, Grant’s depletion experiment suggests it isn’t, and may always be harmful, even if the body tries to use it when it is present. He has depleted his body’s stores as much as possible, but doesn’t seem to have any issues with insufficient immunity to infection/viruses. Perhaps he can speak to the state of his immune system.
If you have evidence that A is needed in some contexts, I’m open minded, but I think most people on this forum are of the opinion that it is never beneficial, and that full elimination is the goal (or as close as reasonably possible).