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Another villain - Tocotrienol?
Quote from lil chick on July 9, 2021, 7:21 amMy own personal opinion is that a mixed diet is best. From a quick search, it appears that it is thought that fish was a big food in ancient egypt, as well as birds (both wild and domestic). Eggs.
I can see, however, that in the cities it could be that the underclass might have been forced to live on the storage foods such as beer and bread and weren't able to hunt and fish. And they might have suffered for the lack of meat over time.
In other google searches, it appears that dairy may have also been a part of the diet, but it isn't agreed how much.
I'm betting there were different diseases if you were a peasant eating beer and bread, than if you were an aristocrat.
I wonder if tocotrienol is present in animal foods the same way that retinol is.
My own personal opinion is that a mixed diet is best. From a quick search, it appears that it is thought that fish was a big food in ancient egypt, as well as birds (both wild and domestic). Eggs.
I can see, however, that in the cities it could be that the underclass might have been forced to live on the storage foods such as beer and bread and weren't able to hunt and fish. And they might have suffered for the lack of meat over time.
In other google searches, it appears that dairy may have also been a part of the diet, but it isn't agreed how much.
I'm betting there were different diseases if you were a peasant eating beer and bread, than if you were an aristocrat.
I wonder if tocotrienol is present in animal foods the same way that retinol is.
Quote from Даниил on July 9, 2021, 8:38 amQuote from lil chick on July 9, 2021, 7:21 amMy own personal opinion is that a mixed diet is best. From a quick search, it appears that it is thought that fish was a big food in ancient egypt, as well as birds (both wild and domestic). Eggs.
I can see, however, that in the cities it could be that the underclass might have been forced to live on the storage foods such as beer and bread and weren't able to hunt and fish. And they might have suffered for the lack of meat over time.
In other google searches, it appears that dairy may have also been a part of the diet, but it isn't agreed how much.
I'm betting there were different diseases if you were a peasant eating beer and bread, than if you were an aristocrat.
I wonder if tocotrienol is present in animal foods the same way that retinol is.
Of course, the Egyptians were quite sophisticated and knew all categories of food. But I don't think they ate them in large quantities (I'm talking about the bulk of the population). Look at what people in poor agricultural countries are eating now. These are mainly grains and beans. The same thing, I think, the Egyptians ate. Something like fish a couple of times a week, eggs once a week, and meat / milk on holidays. The higher castes of course ate more VA.
Quote from lil chick on July 9, 2021, 7:21 amMy own personal opinion is that a mixed diet is best. From a quick search, it appears that it is thought that fish was a big food in ancient egypt, as well as birds (both wild and domestic). Eggs.
I can see, however, that in the cities it could be that the underclass might have been forced to live on the storage foods such as beer and bread and weren't able to hunt and fish. And they might have suffered for the lack of meat over time.
In other google searches, it appears that dairy may have also been a part of the diet, but it isn't agreed how much.
I'm betting there were different diseases if you were a peasant eating beer and bread, than if you were an aristocrat.
I wonder if tocotrienol is present in animal foods the same way that retinol is.
Of course, the Egyptians were quite sophisticated and knew all categories of food. But I don't think they ate them in large quantities (I'm talking about the bulk of the population). Look at what people in poor agricultural countries are eating now. These are mainly grains and beans. The same thing, I think, the Egyptians ate. Something like fish a couple of times a week, eggs once a week, and meat / milk on holidays. The higher castes of course ate more VA.
Quote from Даниил on July 9, 2021, 9:17 amI am also interested to know the content of tocotrienols in meat. I suspect there will be a lot more grain-fed meat. In those studies where nothing was found, grass-fed meat may have been used.
I am also interested to know the content of tocotrienols in meat. I suspect there will be a lot more grain-fed meat. In those studies where nothing was found, grass-fed meat may have been used.
Quote from Даниил on July 11, 2021, 2:36 pmhttps://www.healthbenefitstimes.com/nutrition/gamma-tocotrienol/
I found this site. Here, pork and chicken (I think grain-fed) contain a lot of tocotrienols. Also, fried potatoes and amaranth, unfortunately, have quite high values.
It seems that the only way to get rid of tocotrienols for sure is a diet of grass-fed milk/meat :/
I found this site. Here, pork and chicken (I think grain-fed) contain a lot of tocotrienols. Also, fried potatoes and amaranth, unfortunately, have quite high values.
It seems that the only way to get rid of tocotrienols for sure is a diet of grass-fed milk/meat :/
Quote from Даниил on July 11, 2021, 3:03 pmConditions that (I assume) may be directly or indirectly related to tocotrienols:
tooth sensitivity,
restless legs syndrome,
visceral fat,
fatigue,
bad mood (depression),
nausea,
excessive hairiness,
problems with gaining muscle mass,
If you experience any other symptoms after consuming coconut oil, etc - add to the list
Conditions that (I assume) may be directly or indirectly related to tocotrienols:
tooth sensitivity,
restless legs syndrome,
visceral fat,
fatigue,
bad mood (depression),
nausea,
excessive hairiness,
problems with gaining muscle mass,
If you experience any other symptoms after consuming coconut oil, etc - add to the list
Quote from rockarolla on July 11, 2021, 3:48 pmKeep in mind that coconut oil is very rich in lauric acid(converts to monolaurin) - powerful antibiotic and the symptoms you are describing could be a herxheimer reaction.
https://www.monolaurinandmore.com/articles/monolaurin-die-off-symptoms-the-herxheimer-herx-reaction
I do not handle coconut oil myself well - it adds photosensitivity and neurotoxicity.
Keep in mind that coconut oil is very rich in lauric acid(converts to monolaurin) - powerful antibiotic and the symptoms you are describing could be a herxheimer reaction.
https://www.monolaurinandmore.com/articles/monolaurin-die-off-symptoms-the-herxheimer-herx-reaction
I do not handle coconut oil myself well - it adds photosensitivity and neurotoxicity.
Quote from Даниил on July 14, 2021, 8:39 pmhttps://pubmed.ncbi.nlm.nih.gov/25362611/
"Indiscriminate application of organophosphate (OP) pesticides has led to environmental pollution and severe health problems. The aim of the present study was to evaluate the effect of palm oil tocotrienol-rich fraction (TRF) on biochemical and morphological changes of the liver in rats treated with fenitrothion (FNT), a type of OP pesticide. A total of 28 male Sprague-Dawley rats were divided into four groups; control group, TRF-supplemented group, FNT-treated group and TRF+FNT group. TRF (200 mg/kg) was supplemented 30 minutes prior to FNT (20 mg/kg) administration, both orally for 28 consecutive days. Following 28 days of treatment, plasma biochemical changes and liver morphology were evaluated. The body and absolute liver weights were significantly elevated in TRF+FNT group compared to FNT group. TRF administration significantly decreased the total protein level and restored the activity of alanine aminotransferase (ALT) and aspartate aminotransferase (AST) in TRF + FNT group."
https://pubmed.ncbi.nlm.nih.gov/25362611/
"Indiscriminate application of organophosphate (OP) pesticides has led to environmental pollution and severe health problems. The aim of the present study was to evaluate the effect of palm oil tocotrienol-rich fraction (TRF) on biochemical and morphological changes of the liver in rats treated with fenitrothion (FNT), a type of OP pesticide. A total of 28 male Sprague-Dawley rats were divided into four groups; control group, TRF-supplemented group, FNT-treated group and TRF+FNT group. TRF (200 mg/kg) was supplemented 30 minutes prior to FNT (20 mg/kg) administration, both orally for 28 consecutive days. Following 28 days of treatment, plasma biochemical changes and liver morphology were evaluated. The body and absolute liver weights were significantly elevated in TRF+FNT group compared to FNT group. TRF administration significantly decreased the total protein level and restored the activity of alanine aminotransferase (ALT) and aspartate aminotransferase (AST) in TRF + FNT group."
Quote from Даниил on July 14, 2021, 11:40 pmhttps://pubmed.ncbi.nlm.nih.gov/12353645/
"A tocotrienol (T3) mixture was intragastricaly administered to Sprague-Dawley rats, and the T3 levels in various tissues were measured 0, 4, 8 and 24 hr after the administration. In blood clots, brain, thymus, testes, vice-testes and muscles, T3 homologues were not detected at all. In epididymal adipose, renal adipose, subcutaneous adipose and brown adipose tissues and in the heart, the T3 levels were maintained or increased for 24 hr after the administration. In the serum, liver, mesenteric lymph node, spleen and lungs, the T3 levels were highest 8 hr after the T3 administration. These results suggest that the distribution and metabolism of T3 in the rat vary considerably among different tissues."
I think this data can be extrapolated to other animals. Thus, adipose tissue, blood and animal by-products may contain tocotrienols. Unlike the brain and muscles.
https://pubmed.ncbi.nlm.nih.gov/12353645/
"A tocotrienol (T3) mixture was intragastricaly administered to Sprague-Dawley rats, and the T3 levels in various tissues were measured 0, 4, 8 and 24 hr after the administration. In blood clots, brain, thymus, testes, vice-testes and muscles, T3 homologues were not detected at all. In epididymal adipose, renal adipose, subcutaneous adipose and brown adipose tissues and in the heart, the T3 levels were maintained or increased for 24 hr after the administration. In the serum, liver, mesenteric lymph node, spleen and lungs, the T3 levels were highest 8 hr after the T3 administration. These results suggest that the distribution and metabolism of T3 in the rat vary considerably among different tissues."
I think this data can be extrapolated to other animals. Thus, adipose tissue, blood and animal by-products may contain tocotrienols. Unlike the brain and muscles.
Quote from Даниил on July 15, 2021, 8:28 amI have recently read a lot of studies of tocotrienols and I have the impression that they are full of fraud in order to prove the benefits / hide the harm of tocotrienols. For example, take this study:
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3497010/
Here the mice were given diets with a lot of carbohydrates (corn starch) , a lot of fats, a lot of carbohydrates or fats + tocotrienols. Interestingly, the mice in the high-fat group had huge problems compared to the high-carbohydrate group (just look at the tables) . The fat + tocotrienol group had better health than fat. They did not specify the source of fat in the diet, and I suspect that it was a lot of PUFA, such as flaxseed oil. It is logical that antioxidants, even such as tocotrienols, will protect against PUFA. But I was interested in comparing the high-carbohydrate group with and without tocotrienols (although I think they also had a lot of PUFA in their diet). And, they didn't write it in the review ! Mice with tocotrienols had less muscle mass, more abdominal fat, a higher visceral obesity index, higher cholesterol and a slightly larger liver mass (with a smaller total weight).
In addition, in almost all studies, mice are given tocotrienols with alpha-tocopherol (although it is known that it blocks the adsorption of tocotrienols). And I notice that the higher the percentage of alpha-tocopherol, the better the results.
I have recently read a lot of studies of tocotrienols and I have the impression that they are full of fraud in order to prove the benefits / hide the harm of tocotrienols. For example, take this study:
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3497010/
Here the mice were given diets with a lot of carbohydrates (corn starch) , a lot of fats, a lot of carbohydrates or fats + tocotrienols. Interestingly, the mice in the high-fat group had huge problems compared to the high-carbohydrate group (just look at the tables) . The fat + tocotrienol group had better health than fat. They did not specify the source of fat in the diet, and I suspect that it was a lot of PUFA, such as flaxseed oil. It is logical that antioxidants, even such as tocotrienols, will protect against PUFA. But I was interested in comparing the high-carbohydrate group with and without tocotrienols (although I think they also had a lot of PUFA in their diet). And, they didn't write it in the review ! Mice with tocotrienols had less muscle mass, more abdominal fat, a higher visceral obesity index, higher cholesterol and a slightly larger liver mass (with a smaller total weight).
In addition, in almost all studies, mice are given tocotrienols with alpha-tocopherol (although it is known that it blocks the adsorption of tocotrienols). And I notice that the higher the percentage of alpha-tocopherol, the better the results.
Quote from rockarolla on July 15, 2021, 8:36 amQuote from Daniil on July 15, 2021, 8:28 amI have recently read a lot of studies of tocotrienols and I have the impression that they are full of fraud in order to prove the benefits / hide the harm of tocotrienols.
That's typical almost all research covers supplement/pharma industries profits, sometimes they leak valid info but very rarely.
Quote from Daniil on July 15, 2021, 8:28 amI have recently read a lot of studies of tocotrienols and I have the impression that they are full of fraud in order to prove the benefits / hide the harm of tocotrienols.
That's typical almost all research covers supplement/pharma industries profits, sometimes they leak valid info but very rarely.